Overview of acute coronary syndrome
Esta página compila nosso conteúdo relacionado a acute coronary syndrome. Para obter mais informações sobre o diagnóstico e o tratamento, siga os links abaixo para nossos tópicos completos do BMJ Best Practice sobre as doenças e sintomas relevantes.
Introdução
Condições de saúde relevantes
Unstable angina (UA) | ir para nosso tópico completo sobre Unstable angina (UA) UA is defined as myocardial ischemia at rest or on minimal exertion in the absence of acute cardiomyocyte injury/necrosis.[1] UA is characterized by specific clinical findings of prolonged (>20 minutes) angina at rest; new onset of severe angina; angina that is increasing in frequency, longer in duration, or lower in threshold; or angina that occurs after a recent episode of myocardial infarction.[1] Myocardial biomarkers (no dynamic elevation of cardiac troponin above the 99th percentile) rule out acute myocardial infarction. T-segment depression and T-wave changes may be seen on ECG in patients with UA. Alternatively, the initial ECG may show transient ST elevation, or may be normal.[1] |
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Non-ST-elevation myocardial infarction (NSTEMI) | ir para nosso tópico completo sobre Non-ST-elevation myocardial infarction (NSTEMI) NSTEMI is an acute ischemic event causing irreversible myocyte necrosis. It is usually the result of a transient or near-complete occlusion of a coronary artery or an acute factor that deprives the myocardium of oxygen. NSTEMI is differentiated from UA by a dynamic elevation of troponin above the 99th percentile.[1] Patients with NSTEMI may also be clinically unstable (e.g., low blood pressure, shock, left ventricular failure) which is not a feature of UA. The initial ECG may show ischemic changes such as ST depression, T-wave changes, or transient ST elevation; however, ECG may also be normal or show nonspecific changes. |
ST-elevation myocardial infarction (STEMI) | ir para nosso tópico completo sobre ST-elevation myocardial infarction (STEMI) STEMI is the irreversible necrosis of heart muscle, usually caused by complete atherothrombotic occlusion of a coronary artery. Persistent ST-segment elevation in two or more anatomically contiguous ECG leads is the hallmark ECG pattern. A rise in cardiac-specific troponins confirms the diagnosis. Treatment should, however, be started immediately in patients with a typical history and ECG changes, without waiting for laboratory results.[1] |
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This overview has been compiled using the information in existing sub-topics.
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